Therapeutic and prognosis of IgA nephritis

 1, the supply of protein. Dietary treatment of chronic nephritis should be based on the degree of impairment of renal function to determine the protein intake, if longer duration, renal damage is not serious, you do not have the protein in food strictly limited, but every day should not exceed 1 gram per kilogram of body weight, quality protein to reach 50% or more.
2, due to limitations of the protein part of the patient, and its energy supply should take carbohydrates and fats as the main source of energy supply, as the labor intensity may be. Rest who can supply 126 adult daily kilojoules ~ 147 kJ (30 kcal to 35 kcal) / kg body weight. And activities to meet the needs of the patient.
3, the control of sodium intake. Severe edema and hypertension, to control the amount of sodium in 2 g / day or less, and even give salt-free diet, generally salt is appropriate.
Chronic nephritis food choices:
1, to give plenty of vitamins, especially vitamin c, because chronic nephritis patients may have anemia, vitamin c can increase iron absorption, so should eat tomatoes, green leafy vegetables, fresh dates, watermelon, Radish , cucumber, watermelon, oranges, kiwi fruit and natural juices and other foods.
2, poor appetite may supplement vitamin c preparations; also should add vitamin b and folic acid-rich foods, such as animal offal, green leafy vegetables and other foods, helps to correct anemia. Hyperkalemia should not eat foods high in potassium, should be carefully selected vegetables and fruits. Chronic nephritis patients should not eat sugar drinks and spicy foods.
Prognosis:
The disease may have spontaneous remission, or about 4% to 20%. Each year, about 1% to 2% of the cases into end-stage renal failure. Life-table analysis showed that 10-year statistical graft survival was 80% to 90%. Estimated that eventually developed into uremia approximately 35%. The rest is persistent hematuria or proteinuria. Poor prognosis factors: the onset of renal dysfunction, proteinuria exceeding 1.5g / d, high blood pressure and no gross hematuria; Renal glomerular sclerosis, interstitial fibrosis, glomerular capillary is violations, diffuse hyperplasia and diffuse crescent formation.